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Universitätsstr. 5
45141 Essen
45141 Essen
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S05 T04 B23
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Die folgenden Publikationen sind in der Online-Universitätsbibliographie der Universität Duisburg-Essen verzeichnet. Weitere Informationen finden Sie gegebenenfalls auch auf den persönlichen Webseiten der Person.
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Anthracycline Therapy Modifies Immune Checkpoint Signaling in the HeartIn: International Journal of Molecular Sciences (IJMS) Jg. 24 (2023) Nr. 7, 6052Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Targeting early stages of cardiotoxicity from anti-PD1 immune checkpoint inhibitor therapyIn: European Heart Journal Jg. 43 (2022) Nr. 4, S. 316 - 329Online Volltext: dx.doi.org/
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Superiority of focused ion beam-scanning electron microscope tomography of cardiomyocytes over standard 2D analyses highlighted by unmasking mitochondrial heterogeneityIn: Journal of Cachexia, Sarcopenia and Muscle Jg. 12 (2021) Nr. 4, S. 933 - 954Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Synergistic activity of BET inhibitor MK-8628 and PLK inhibitor Volasertib in preclinical models of medulloblastomaIn: Cancer Letters Jg. 455 (2019) S. 24 - 33Online Volltext: dx.doi.org/
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RITA displays anti-tumor activity in medulloblastomas independent of TP53 statusIn: OncoTarget Jg. 8 (2017) Nr. 17, S. 27882 - 27891Online Volltext: dx.doi.org/ (Open Access)
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Targeting tachykinin receptors in neuroblastomaIn: OncoTarget Jg. 8 (2017) Nr. 1, S. 430 - 443Online Volltext: dx.doi.org/ (Open Access)
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The GSK461364 PLK1 inhibitor exhibits strong antitumoral activity in preclinical neuroblastoma modelsIn: OncoTarget Jg. 8 (2017) Nr. 4, S. 6730 - 6741Online Volltext: dx.doi.org/ (Open Access)
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Targeting MYCN-driven transcription by BET-bromodomain inhibitionIn: Clinical Cancer Research Jg. 22 (2016) Nr. 10, S. 2470 - 2781Online Volltext: dx.doi.org/ (Open Access)
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Absence of telomerase reverse transcriptase promoter mutations in neuroblastomaIn: Biomedical Reports Jg. 3 (2015) Nr. 4, S. 443 - 446Online Volltext: dx.doi.org/
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Mutational dynamics between primary and relapse neuroblastomasIn: Nature Genetics Jg. 47 (2015) Nr. 8, S. 872 - 877Online Volltext: dx.doi.org/
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Targeting super-enhancer induced gene expression with the novel BRD4 inhibitor OTX015 in preclinical models of MYCN-amplified neuroblastomaIn: Cancer Research Jg. 75 (2015) Nr. 15 Suppl., S. 4731Online Volltext: dx.doi.org/
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miR-542-3p exerts tumor suppressive functions in neuroblastoma by downregulating SurvivinIn: International Journal of Cancer Jg. 136 (2015) Nr. 6, S. 1308 - 1320Online Volltext: dx.doi.org/ (Open Access)
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BET protein inhibitor OTX015 has selective anti-tumoral activity in preclinical models of MYCN- amplified neuroblastomaIn: Cancer Research Jg. 74 (2014) Nr. 19 Suppl., S. 3967Online Volltext: dx.doi.org/
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Neuroblastoma in dialog with its stroma : NTRK1 is a regulator of cellular cross-talk with Schwann cellsIn: OncoTarget Jg. 5 (2014) Nr. 22, S. 11180 - 11192Online Volltext: dx.doi.org/ (Open Access)
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BET bromodomain protein inhibition is a therapeutic option for medulloblastomaIn: OncoTarget Jg. 4 (2013) Nr. 11, S. 2080 - 2095Online Volltext: dx.doi.org/
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MiR-137 functions as a tumor suppressor in neuroblastoma by downregulating KDM1AIn: International Journal of Cancer Jg. 133 (2013) Nr. 5, S. 1064 - 1073Online Volltext: dx.doi.org/
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Targeted expression of mutated ALK induces neuroblastoma in transgenic miceIn: Science Translational Medicine Jg. 4 (2012) Nr. 141, S. 141ra91Online Volltext: dx.doi.org/
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Pharmacologic activation of the p53 pathway using Nutlin-3 inhibits medulloblastoma cell proliferation in vitroIn: Klinische Pädiatrie Jg. 221 (2009) Nr. 3, S. 201Online Volltext: dx.doi.org/
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The mutagenic potential of non-homologous end joining in the absence of the NHEJ core factors Ku70/80, DNA-PKcs and XRCC4-LigIVIn: Mutagenesis Jg. 22 (2007) Nr. 3, S. 217 - 233Online Volltext: dx.doi.org/
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Analysis of double-strand break repair by nonhomologous DNA end joining in cell-free extracts from mammalian cellsIn: Molecular Toxicology Protocols / Keohavong, Phouthone; Grant, Stephen G. (Hrsg.) 2014, S. 565 - 585Online Volltext: dx.doi.org/